Staff Publications

Staff Publications

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    'Staff publications' is the digital repository of Wageningen University & Research

    'Staff publications' contains references to publications authored by Wageningen University staff from 1976 onward.

    Publications authored by the staff of the Research Institutes are available from 1995 onwards.

    Full text documents are added when available. The database is updated daily and currently holds about 240,000 items, of which 72,000 in open access.

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Profiling healthy eaters. Determining factors that predict healthy eating practices among Dutch adults
Swan, E.C. ; Bouwman, L.I. ; Hiddink, G.J. ; Aarts, N. ; Koelen, M. - \ 2015
Appetite 89 (2015). - ISSN 0195-6663 - p. 122 - 130.
coronary-heart-disease - life-style choices - socioeconomic inequalities - physical-activity - dietary patterns - social support - public-health - food choice - weight-gain - us adults
Research has identified multiple factors that predict unhealthy eating practices. However what remains poorly understood are factors that promote healthy eating practices. This study aimed to determine a set of factors that represent a profile of healthy eaters. This research applied Antonovsky's salutogenic framework for health development to examine a set of factors that predict healthy eating in a cross-sectional study of Dutch adults. Data were analyzed from participants (n¿=¿703) who completed the study's survey in January 2013. Logistic regression analysis was performed to test the association of survey factors on the outcome variable high dietary score. In the multivariate logistic regression model, five factors contributed significantly (p¿
Joint Association of Dietary Pattern and Physical Activity Level with Cardiovascular Disease Risk Factors among Chinese Men: A Cross-Sectional Study
Wang, D. ; He, Y. ; Li, Y.P. ; Luan, D.C. ; Zhai, F.Y. ; Yang, X.G. ; Ma, G.S. - \ 2013
PLoS One 8 (2013)6. - ISSN 1932-6203
diabetes prevention program - impaired glucose-tolerance - life-style - metabolic syndrome - blood-pressure - attributable risk - plasma biomarkers - us adults - population - hypertension
The purpose of this cross-sectional study was to investigate the joint associations of physical activity level (PAL) and dietary patterns in relation to cardiovascular disease (CVD) risk factors among Chinese men. The study population consisted of 13 511 Chinese males aged 18-59 years from the 2002 China National Nutrition and Health Survey. Based on dietary data collected by a food frequency questionnaire, four dietary patterns were identified and labeled as "Green Water'' (high consumption of rice, vegetables, seafood, pork, and poultry), "Yellow Earth'' (high consumption of wheat flour products and starchy tubers), "New Affluent'' (high consumption of animal sourced foods and soybean products), and "Western Adopter'' (high consumption of animal sourced foods, cakes, and soft drinks). From the information collected by a 1-year physical activity questionnaire, PAL was calculated and classified into 4 categories: sedentary, low active, active, and very active. As compared with their counterparts from the New Affluent pattern, participants who followed the Green Water pattern had a lower likelihood of abdominal obesity (AO; 50.2%), hypertension (HT; 37.9%), hyperglycemia (HG; 41.5%), elevated triglyceride (ETG; 14.5%), low HDL (LHDL; 39.8%), and metabolic syndrome (MS; 51.9%). When compared to sedentary participants, the odds ratio of participants with very active PAL was 0.62 for AO, 0.85 for HT, 0.71 for HG, 0.76 for ETG, 0.74 for LHDL, and 0.58 for MS. Individuals who followed both very active PAL and the Green Water pattern had a lower likelihood of CVD risk factors (AO: 65.8%, HT: 39.1%, HG: 57.4%, ETG: 35.4%, LHDL: 56.1%, and MS: 75.0%), compared to their counterparts who followed both sedentary PAL and the New Affluent pattern. In addition, adherence to both healthy dietary pattern and very active PAL presented a remarkable potential for CVD risk factor prevention.
The association between dietary energy density and type 2 diabetes in Europe: results form the EPIC-InterAct Study
The InterAct Consortium, A. ; Groenendijk-van Woudenbergh, G.J. ; Feskens, E.J.M. - \ 2013
PLoS One 8 (2013)5. - ISSN 1932-6203 - 10 p.
impaired glucose-tolerance - life-style - metabolic syndrome - glycemic load - united-states - weight change - us adults - risk - women - mellitus
Background Observational studies implicate higher dietary energy density (DED) as a potential risk factor for weight gain and obesity. It has been hypothesized that DED may also be associated with risk of type 2 diabetes (T2D), but limited evidence exists. Therefore, we investigated the association between DED and risk of T2D in a large prospective study with heterogeneity of dietary intake. Methodology/Principal Findings A case-cohort study was nested within the European Prospective Investigation into Cancer (EPIC) study of 340,234 participants contributing 3.99 million person years of follow-up, identifying 12,403 incident diabetes cases and a random subcohort of 16,835 individuals from 8 European countries. DED was calculated as energy (kcal) from foods (except beverages) divided by the weight (gram) of foods estimated from dietary questionnaires. Prentice-weighted Cox proportional hazard regression models were fitted by country. Risk estimates were pooled by random effects meta-analysis and heterogeneity was evaluated. Estimated mean (sd) DED was 1.5 (0.3) kcal/g among cases and subcohort members, varying across countries (range 1.4–1.7 kcal/g). After adjustment for age, sex, smoking, physical activity, alcohol intake, energy intake from beverages and misreporting of dietary intake, no association was observed between DED and T2D (HR 1.02 (95% CI: 0.93–1.13), which was consistent across countries (I2 = 2.9%). Conclusions/Significance In this large European case-cohort study no association between DED of solid and semi-solid foods and risk of T2D was observed. However, despite the fact that there currently is no conclusive evidence for an association between DED and T2DM risk, choosing low energy dense foods should be promoted as they support current WHO recommendations to prevent chronic diseases.
Serum 25-hydroxyvitamin D is associated with cognitive executive function in Dutch prefrail and frail elderly: a cross-sectional study exploring the associations of 25-hydroxyvitamin D with glucose metabolism, cognitive performance and depression
Brouwer, E.M. ; Nieuwerth-van de Rest, O. ; Tieland, C.A.B. ; Zwaluw, N.L. van der; Steegenga, W.T. ; Adam, J.J. ; Loon, L.J.C. van; Feskens, E.J.M. ; Groot, C.P.G.M. de - \ 2013
Journal of the American Medical Directors Association 14 (2013)1. - ISSN 1525-8610 - p. 852.e9 - 852.e17.
randomized controlled-trial - vitamin-d supplementation - placebo-controlled trial - parathyroid-hormone - insulin-resistance - double-blind - older women - risk-factors - us adults - population
Objectives: The primary objective was to explore the possible association of serum 25-hydroxyvitamin D (25[OH] D) and vitamin D intake with markers of glucose metabolism, depression, and cognitive performance. In addition, we examined to what extent the associations between vitamin D and cognitive performance were modified or mediated by fasting plasma glucose (FPG) levels. Design, Setting, and Participants: Cross-sectional study using data of 127 frail or prefrail Dutch elderly, aged 65 years or older. Frailty was defined according to the criteria of Fried and colleagues. A participant was classified prefrail when 1 to 2 criteria were met; frailty was classified as the presence of 3 or more criteria. Measurements: Associations of 25(OH) D and vitamin D intake with markers of glucose metabolism and domain-specific cognitive performance were examined by multivariable regression analyses. The possible association of vitamin D with depression and global cognitive performance was explored by Poisson regression. Results: No associations were observed for 25(OH) D with FPG, fasting plasma insulin (FPI), Homeostasis Model Assessment-estimated Insulin Resistance (HOMA-IR), or depression. In contrast, serum 25(OH) D was positively associated with executive functioning (beta 0.007, P=.01) and tended to be associated with information-processing speed (beta 0.006, P=.06). FPG did not modify or mediate these associations. Vitamin D intake was not associated with cognitive performance, glucose metabolism, or depression. Conclusion: This cross-sectional study suggests an association of serum 25(OH) D with domain-specific cognitive performance, in particular executive functioning and possibly information-processing speed, but not with FPG, FPI, HOMA-IR, or depression. Whether these associations are causal is yet to be demonstrated. Copyright (C) 2013 - American Medical Directors Association, Inc.
A diet high in resistant starch modulates microbiota composition, SCFA concentrations, and gene expression in pig intestine
Haenen, D. ; Zhang, J. ; Souza Da Silva, C. ; Bosch, G. ; Meer, I.M. van der; Arkel, J. van; Borne, J.J.G.C. van den; Pérez Gutiérrez, O. ; Smidt, H. ; Kemp, B. ; Müller, M.R. ; Hooiveld, G.J.E.J. - \ 2013
The Journal of Nutrition 143 (2013)3. - ISSN 0022-3166 - p. 274 - 283.
chain fatty-acids - glucagon-like peptide-1 - phylogenetic microarray - gastrointestinal-tract - human gut - appetite regulation - metabolic syndrome - colonic function - us adults - body-fat
Resistant starch (RS) is highly fermentable by microbiota in the colon, resulting in the production of SCFAs. RS is thought to mediate a large proportion of its health benefits, including increased satiety, through the actions of SCFAs. The aim of this study was to investigate the effects of a diet high in RS on luminal microbiota composition, luminal SCFA concentrations, and the expression of host genes involved in SCFA uptake, SCFA signaling, and satiety regulation in mucosal tissue obtained from small intestine, cecum, and colon. Twenty adult female pigs were either assigned to a digestible starch (DS) diet or a diet high in RS (34%) for a period of 2 wk. After the intervention, luminal content and mucosal scrapings were obtained for detailed molecular analysis. RS was completely degraded in the cecum. In both the cecum and colon, differences in microbiota composition were observed between DS- and RS-fed pigs. In the colon these included the stimulation of the healthy gut-associated butyrate-producing Faecalibacterium prausnitzii, whereas potentially pathogenic members of the Gammaproteobacteria, including Escherichia coli and Pseudomonas spp., were reduced in relative abundance. Cecal and colonic SCFA concentrations were significantly greater in RS-fed pigs, and cecal gene expression of monocarboxylate transporter 1 (SLC16A1) and glucagon (GCG) was induced by RS. In conclusion, our data show that RS modulates microbiota composition, SCFA concentrations, and host gene expression in pig intestine. Combined, our data provide an enhanced understanding of the interaction between diet, microbiota, and host
Relationship between body mass index and mortality among Europeans
Song, X. ; Pitkaniemi, J. ; Heine, R.J. ; Pyorala, K. ; Soderberg, S. ; Stehouwer, C.D. ; Feskens, E.J.M. - \ 2012
European Journal of Clinical Nutrition 66 (2012)2. - ISSN 0954-3007 - p. 156 - 165.
coronary-heart-disease - middle-aged men - follow-up - us adults - cardiovascular risk - original whitehall - physical-activity - obesity - overweight - women
Background/Objectives: To investigate the relationship between body mass index (BMI) and mortality from various causes. Subjects/Methods: Data of 72¿947 European men and 62¿798 women aged 24–99 years at baseline were collaboratively analyzed. Both absolute and relative mortality risks were estimated within each BMI categories. The hazard ratio was estimated using Cox regression analysis adjusting for age, cohort and smoking status. Results: Over a median follow-up of 16.8 years, 29¿071 participants died, 13¿502 from cardiovascular disease (CVD) and 8748 from cancers of all types. All-cause and cancer mortality showed a U-shaped relationship: decreased first, leveled off, and then increased with increasing BMI with the lowest mortality risk approximately between 23.0 and 28.0¿kg/m2 of BMI in men and 21.0 and 28.0¿kg/m2 in women. The U-shaped relationship held for all-cause mortality but disappeared for cancer mortality among non-smokers. The CVD mortality was constant until a BMI of approximately 28.0¿kg/m2 and then increased gradually in both men and women, which was independent of age, cohort and smoking status. Conclusions: A U-shaped relationship of BMI with all-cause mortality but a graded relationship with CVD mortality at BMI >28.0¿kg/m2 was detected. The relationship between cancer mortality and BMI largely depended on smoking status, and need to be further investigated with site-specific cancers
“Infectobesity: viral infections (especially with human adenovirus-36: Ad-36) may be a cause of obesity
Ginneken, V.J.T. van; Sitnyakowsky, L. ; Jeffery, J.E. - \ 2009
Medical Hypotheses 72 (2009)4. - ISSN 0306-9877 - p. 383 - 388.
canine-distemper virus - metabolic syndrome - national-health - us adults - prevalence - overweight - adiposity - disease - model - preadipocytes
In recent years viral infections have been recognized as possible cause of obesity, alongside the traditionally recognized causes (genetic inheritance, and behaviour/environmental causes such as diet exercise, cultural practices and stress). Although four viruses have been reported to induce obesity (infectoobesity) in animal models (chickens, mice, sheep, goat, dogs, rats and hamsters), until recently the viral etiology of human obesity has not received sufficient attention, possibly because the four viruses are not able to infect humans. In a series of papers over the last ten years, however, the group of Prof. Dhurandhar (Pennington Biomedical Research Center, LA, USA) demonstrated that a human adenovirus, adenovirus-36 (Ad-36), is capable of inducing adiposity in experimentally infected chickens, mice and non-human primates (marmosets). Ad-36 is known to increase the replication, differentiation, lipid accumulation and insulin sensitivity in fat cells and reduces those cells’ leptin secretion and expression. It also affects human primary preadipocytes. In rats increased adiposity was observed due to Ad-36 infection. Recent studies have shown that, in the USA, antibodies to Ad-36 were more prevalent in obese subjects (30%) than in non-obese subjects (11%). We postulate that Ad-36 may be a contributing factor to the worldwide rising problem of obesity. We suggest the extension of comparative virological studies between North America and Europe, and studies between discordant twins (both dizygous and monozygous)
Metabolic syndrome and incidence of type 2 diabetes in patients with manifest vascular disease
Wassink, A.M.J. ; Graaf, Y. van der; Soedamah-Muthu, S.S. ; Spiering, W. ; Visseren, F.L.J. - \ 2008
Diabetes & Vascular Disease Research 5 (2008)2. - ISSN 1479-1641 - p. 114 - 122.
coronary-heart-disease - treatment panel-iii - prior myocardial-infarction - 10-year follow-up - cardiovascular-disease - insulin-resistance - nondiabetic subjects - fasting glucose - risk-factors - us adults
Risk reduction in patients with clinically manifest vascular disease focuses on preventing new vascular events and not on prevention of type 2 diabetes. However, given the common pathophysiological pathways involved in the development of atherosclerosis and type 2 diabetes, it is probable that people with atherosclerotic vascular disease have an elevated risk of type 2 diabetes. The present prospective cohort study investigated the incidence of type 2 diabetes and the effect of the presence of metabolic syndrome on the incidence of type 2 diabetes in 4,022 patients with clinically manifest atherosclerosis, included in the study from September 1996 to June 2006. Patients who died (n=456), who were lost to follow-up (n=84) and those with diabetes at baseline (n=558) were excluded, leaving 2,924 patients for analysis. The incidence of diabetes was assessed by questionnaire (self-reported diabetes). During 13,726 person-years of follow-up (median follow-up 4.3 years, range 2.4-7.0 years), there were 152 type 2 diabetes cases (5.2%), corresponding to an incidence rate of 11.1 (95% CI 9.4-13.0) per 1,000 person-years. Patients with metabolic syndrome were at increased risk of incident type 2 diabetes compared to those without metabolic syndrome, with an adjusted hazard ratio of 5.7 (95% CI 3.7-8.9) for Revised National Cholesterol Education Program, 6.0 (4.1-9.0) for National Cholesterol Education Program and 4.0 (2.7-6.1) for International Diabetes Federation definitions of metabolic syndrome. Of all metabolic syndrome components, abdominal obesity was most strongly associated with incident type 2 diabetes (94% higher risk of type 2 diabetes for 1 standard deviation (11.3 cm) increase in waist circumference). In conclusion, patients with manifest atherosclerosis are at high risk of developing type 2 diabetes. Metabolic syndrome identifies those at the highest risk and is an easy to use clinical tool. Abdominal obesity is a strong individual predictor of type 2 diabetes. Patients with manifest atherosclerosis and metabolic syndrome may derive particular benefit from lifestyle interventions focusing on weight reduction.
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